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Jesse H
Mitochondrial glutathione depletion functions not as a downstream marker of oxidative stress, but as a central driver of disease amplification
13a, Supplementary Table 9), suggesting that the distinct rotamer orientation of H296 ECL2 in Cagri-bound CTR (to accommodate the Glu linker) and/or the observed more open ECL3 conformation induced by the lipidated peptide may facilitate or stabilize an active CTR conformation (Supplementary Fig
Adequate intake of cysteine and NAC is vital for health as they help replenish glutathione, the most potent antioxidant in the body
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